Gastroenterology· Topic 5.10 Cholestatic, inherited, and other liver disease· 5.10.5

Wilson disease

Wilson disease is an autosomal recessive disorder of the ATP7B copper-transporting ATPase causing toxic copper accumulation in the liver, brain, and cornea, presenting with hepatic and/or neuropsychiatric disease usually before age 40; diagnosis rests on ceruloplasmin, urinary copper, and Kayser-Fleischer rings, and lifelong treatment is chelation with penicillamine or trientine, or zinc, unless liver transplant is required.

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What this article covers

  • Overview

    Definition: Wilson disease (hepatolenticular degeneration) is an autosomal recessive disorder of copper metabolism in which a defective hepatocyte copper-transporting ATPase causes toxic copper…

  • Epidemiology

    Onset typically before age 40, with hepatic presentations skewing toward children and adolescents and neurologic presentations skewing toward older patients within that range.

  • Risk factors

    Family history of Wilson disease, especially an affected sibling, given autosomal recessive inheritance.

  • Pathophysiology

    Mutation of the copper-transporting ATPase gene (ATP7B) impairs two linked hepatocyte functions: incorporation of copper into apoceruloplasmin to form ceruloplasmin, and biliary excretion of excess…

  • Clinical features

    Hepatic manifestations, more common in younger patients: range from asymptomatic transaminase elevation and chronic hepatitis to steatohepatitis, insidiously developing cirrhosis, and acute liver…

  • Differential diagnosis

    Hemochromatosis: another metal storage disease presenting with cirrhosis, but with iron overload rather than copper overload, elevated serum iron and ferritin with markedly elevated transferrin…

  • Diagnosis

    The diagnosis is confirmed by a low serum ceruloplasmin level (typically below 20 mg/dL) together with an increased urinary or hepatic copper concentration (24-hour urinary copper typically above 100…

  • Management

    First, confirm the diagnosis and start lifelong copper-lowering treatment promptly, since Wilson disease is progressive and largely reversible if started before advanced organ damage occurs.

  • Complications

    Cirrhosis and its sequelae, including portal hypertension, ascites, and variceal bleeding, in patients with unrecognized or inadequately treated hepatic disease.

  • Prognosis

    Excellent prognosis with early diagnosis and adherence to lifelong therapy, particularly when treatment is started before significant hepatic or neurologic damage has occurred.

  • Prevention

    Screen all first-degree relatives, especially siblings, of every confirmed patient for Wilson disease with serum ceruloplasmin (looking for a value below 20 mg/dL), liver function testing, and…

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